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Dr. Ying Qin, Ph.D.

Researcher

Present

Dongguan Institute of Reproductive and Genetic Research

Dongguan

China

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I graduated from Xiangya Medical College, Central South University in China with a bachelor's degree. Then, I graduated with a PhD in Medicine from Nagoya University Graduate School in 2019. My research direction is related to premature ovarian failure and polycystic ovary syndrome in reproductive medicine. I worked at the Obstetrics and Gynecology and Reproductive Center of Guangzhou Women and Children's Medical Center from 2019 to 2024, while also conducting research in the field of reproductive medicine. I have been working as an attending physician at the reproductive center of Dongguan Maternal and Child Health Hospital and a researcher of Dongguan Institute of Reproductive and Genetic Research since 2025.

  • Premature ovarian failure
  • Polycystic ovary syndrome
  • Stem cell
  • Reproductive Endocrinology
  • Reproductive immunity
  • Assisted reproduction
  • Endometriosis
  • Qin Y, Iwase A, Murase T, Bayasula, Ishida C, Kato N, Nakamura T, Osuka S, Takikawa S, Goto M, Kotani T, Kikkawa F. Protective effects of mangafodipir against chemotherapy-induced ovarian damage in mice. Reprod Biol Endocrinol. 2018 Oct 27;16(1):106.
  • Qin Y, Wen C, Hu B, Wu H. Investigating the potential role of α-SNAP in preventing chemotherapy-induced ovarian dysfunction: Insights from cellular and animal models. Heliyon. 2024 Jun 10;10(12):e32802.
  • Qin Y, Wen C, Wu H. CXCL10-based gene cluster model serves as a potential diagnostic biomarker for premature ovarian failure. PeerJ. 2023 Dec 13;11:e16659.
  • Qin Y. Effects of using letrozole in combination with the GnRH antagonist protocol for patients with poor ovarian response: A meta-analysis. J Gynecol Obstet Hum Reprod. 2021 Oct;50(8):102139.
  • Kato N, Iwase A, Ishida C, Nagai T, Mori M, Bayasula, Nakamura T, Osuka S, Ganiyeva U, Qin Y, Miki R, Kikkawa F. Upregulation of Fibroblast Growth Factors Caused by Heart and Neural Crest Derivatives Expressed 2 Suppression in Endometriotic Cells: A Possible Therapeutic Target in Endometriosis. Reprod Sci. 2019 Jul;26(7):979-987.
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